But they’re poisonous! You say. And that’s true, you absolutely should not eat raw amanitas. But you know what other mushrooms you shouldn’t eat raw? All of them! Chitin is indigestible!

“Well, sure, but a raw porcini won’t kill me!” That’s true, but you know what will? A Morel! (If you’re small). At the very least they’ll make you as sick as an amanita. And you know what people do with Morels? They eat them! They’re a prize edible!

“Okay, but people eat morrells but they don’t eat Amanitas” okay. Well, take off your fucking anglocentrism glasses because they’ve been eating amanitas in India and Japan since before your country even existed.

So listen up, slice and boil your amanitas in gallons of water, and I mean gallons. Strain and rinse. Boil, strain and rinse again. Guess what you’ve got? A soft, buttery, delicious mushroom. Pour some garlic butter on it, or do it authentic and put it into miso soup as a nice earthy addition.

It’s an edible. Change my mind.

  • BigHaas [he/him]@hexbear.netEnglish
    4·
    1 year ago

    Not a deliriant, it’s a weird gabaergic like Ambien

    https://m.psychonautwiki.org/wiki/Amanita_muscaria Muscimol has an atypical mechanism of action. Unlike classical psychedelics (which are 5-HT2A agonists) and classical dissociatives (which are NMDA receptor antagonists), muscimol is a potent GABAA agonist, meaning it activates the receptor for GABA, the brain’s principle inhibitory neurotransmitter. As an agonist, muscimol binds to the same site on the GABAA receptor as GABA itself. This is unlike benzodiazepines and barbiturates, which bind to different allosteric sites on the GABAA receptor.

    Muscimol has also been shown to be a partial agonist at the GABAA-ρ receptor, which may contribute to its psychoactive effects. Activation of α1 subunit GABA receptors is responsible for the hallucinogenic effects of certain GABAergic compounds such as z-drugs, including zolpidem, and muscimol.[7]

    The effects of isolated muscimol begin approximately one hour after consumption, peaking at 3 hours and lasting a total of 10-24 hours.[8]

    Ibotenic acid has been shown to be a potent NMDA agonist as well as a powerful agonist at the group I and group II metabotropic glutamate receptors. Due to in vivo decarboxylation, ibotenic acid is metabolized to muscimol and thus has many similar pharmacological characteristics